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khad
22nd August 2011, 02:22
While this new booming field does not invalidate Darwin, it's interesting because it shows that environmental factors and acquired factors may unlock genetic "switches" that lead to different characteristics. For so long epigenetic research has been stigmatized as debunked trash, but now researchers are coming up with concrete results that show a much more complicated picture of genetic interaction.


http://www.sciencedaily.com/releases/2009/04/090412081315.htm


. Epigenetics: DNA Isn’t Everything

http://images.sciencedaily.com/2009/04/090412081315.jpg (http://images.sciencedaily.com/2009/04/090412081315-large.jpg)
The two pictures show the eyes of two genetically identical flies. The difference in eye colour is determined by epigenetic factors. (Credit: Renato Paro/ETH Zürich)


ScienceDaily (Apr. 13, 2009) — Research into epigenetics has shown that environmental factors affect characteristics of organisms. These changes are sometimes passed on to the offspring. ETH professor Renato Paro does not believe that this opposes Darwin’s theory of evolution.

A certain laboratory strain of the fruit fly Drosophila melanogaster has white eyes. If the surrounding temperature of the embryos, which are normally nurtured at 25 degrees Celsius, is briefly raised to 37 degrees Celsius, the flies later hatch with red eyes. If these flies are again crossed, the following generations are partly red-eyed – without further temperature treatment – even though only white-eyed flies are expected according to the rules of genetics.

Environment affects inheritance

Researchers in a group led by Renato Paro, professor for Biosystems at the Department of Biosystems Science and Engineering (D-BSSE), crossed the flies for six generations. In this experiment, they were able to prove that the temperature treatment changes the eye colour of this specific strain of fly, and that the treated individual flies pass on the change to their offspring over several generations. However, the DNA sequence for the gene responsible for eye colour was proven to remain the same for white-eyed parents and red-eyed offspring.

The concept of epigenetics offers an explanation for this result. Epigenetics examines the inheritance of characteristics that are not set out in the DNA sequence. For Paro, epigenetic mechanisms form an additional, paramount level of information to the genetic information of DNA.

Such phenomena could only be examined in a descriptive manner in the past. Today, it has been scientifically proven, which molecular structures are involved: important factors are the histones, a kind of packaging material for the DNA, in order to store DNA in an ordered and space-saving way. It is now clear that these proteins have additional roles to play. Depending on the chemical group they carry, if they are acetylated or methylated, they permanently activate or deactivate genes. New methods now allow researchers to sometimes directly show which genes have been activated or deactivated by the histones.

Cells have a memory

Epigenetic marks, such as the modifications of the histones, are also important for the specialisation of the body’s cells. They are preserved during cell division and are passed on to the daughter cells. If skin cells divide, more skin cells are created; liver cells form liver cells. In both cell types, all genes are deactivated except the ones needed by a skin or liver cell to be a skin or liver cell, and to function appropriately. The genetic information of the DNA is passed on along with the relevant epigenetic information for the respective cell type.

Paro’s group is researching this cell memory. It is still unclear how the epigenetic markers are passed on to the daughter cells. During cell division, the DNA is doubled, which requires the histones – as the current picture suggests – to break apart. The question is therefore how cellular memory encoded by epigenetic mechanisms survives cell division.

Emerging area of research

A similar question remains for the inheritance of the epigenetic characteristics from parents to offspring. They now know that when the gametes are formed, certain epigenetic markers remain and are passed on to the offspring. The questions, which are currently being researched, are how much and which part of the epigenetic information is preserved and subsequently inherited.

The research is also looking at the influence of various substances from the environment on the epigenetic constitution of organisms, including humans. Diet and epigenetics appear to be closely linked. The most well known example is that of the Agouti mice: they are yellow, fat and are prone to diabetes and cancer. If Agouti females are fed with a cocktail of vitamin B12, folic acid and cholin, directly prior to and during pregnancy, they give birth to mainly brown, slim and healthy offspring. They in turn mainly have offspring similar to themselves.

Contradiction to Darwin?

Environmental factors, which change the characteristics of an individual and are then passed on to its offspring, do not really fit into Darwin’s theory of evolution. According to his theory, evolution is the result of the population and not the single individual. “Passing on the gained characteristics fits more to Lamarck’s theory of evolution”, says Paro.

However, he still does not believe Darwin’s theory of evolution is put into question by the evidence of epigenetics research. “Darwin was 100 percent right”, Paro emphasises. For him, epigenetics complement Darwin’s theory. In his view, new characteristics are generated and passed on via epigenetics, subject to the same mechanisms of evolution as those with a purely genetic origin.http://www.thedailybeast.com/newsweek/2009/09/17/what-alters-our-genes.html


What Alters Our Genes
Sep 17, 2009 8:00 PM EDT

For a time, it was the most famous fraud in biology. From 1906 to 1923, Austrian biologist Paul Kammerer reported remarkable results in experiments with the midwife toad. Highly unusual for an amphibian, Alytes obstetricans mates on land, not water, and the males incubate the eggs on their legs, also on land. But when Kammerer housed midwife toads in a hot, dry terrarium, they spent most of their time in a nearby basin of cool water: they mated there, and mom deposited the eggs there rather than having dad carry them. It was odd enough that so intrinsic (and eponymous) a behavior was so malleable. But Kammerer found something else.

Once the tadpoles grew up—and here is where eyebrows all over European science shot up—they mated and deposited their eggs in water. Dads no longer midwifed eggs, even when they did not live in desert conditions. By the fourth generation, Kammerer reported, male toads even had black "nuptial" pads, a trait water-mating toads have (to grasp a female) but midwife toads do not. Thus did Kammerer demonstrate the inheritance of acquired characteristics, the then- and still-discredited idea that a new behavioral trait (such as reproducing in water) or anatomical one (nuptial pads) can be environmentally induced and passed on to progeny, despite the progeny's never having been exposed to that environment.

Other scientists were dubious. This was before the discovery that DNA is the molecule of heredity, but basic Darwinian and Mendelian theory held that traits are inherited intact from mom and dad, and that the experiences a parent has cannot alter the genetic material in eggs and sperm. Indeed, one of Kammerer's critics found that India ink had been injected into a toad, creating faux nuptial pads; on Aug. 7, 1926, a paper in Nature suggested that Kammerer had committed fraud. Six weeks later, he committed suicide. Arthur Koestler made the saga the subject of his 1971 book, The Case of the Midwife Toad.

Despite attempts here and there to restore his reputation, Kammerer's name became synonymous with science fraud. But in a fascinating new analysis, biologist Alexander Vargas of the University of Chile reaches a far different conclusion: that Kammerer was in fact the discoverer of a phenome-non called epigenetics, in which genes are silenced by—and here I'm simplifying, but only a little—experience. "Rather than being a fraud," says Vargas, "Kammerer could be the true discoverer of non-Mendelian, epigenetic inheritance."

Epigenetics is a booming new field that studies how genes are turned on and off. At the molecular level, that can happen when a cluster of four atoms called a methyl group attaches to a gene, silencing it. What's so fascinating about epigenetics is that it may explain how the life we live can reach into our double helix and alter our traits. For instance, when rat moms lick and groom their pups, it removes DNA silencers from genes that, allowed to sing, cause the pups to become curious and sociable. Epigenetic mechanisms may also explain why identical twins, who inherit identical genes, have different traits, including genetic diseases: the different lives the twins lead cause some disease genes, including those linked to cancer and schizophrenia, to switch off.

It turns out that when eggs, including those of amphibians, spend time in water—as did those of Kammerer's midwife toads—the DNA within undergoes a wave of methylation, turning some genes on and others off. If one such set of genes carries instructions for living on land and midwifing eggs, then turning it off would cause the toad to revert to a classically amphibian way of life, reproducing in water, as Kammerer found. Genes for living on land seem to get "environmentally silenced in early embryos exposed to water," says Vargas, who combed through Kammerer's lab notes and whose analysis appears in the Journal of Experimental Zoology. "It has taken a painfully long time to properly acknowledge that environment can influence inheritance," he told me. "I think academia has discouraged experiments testing environmental modification of inheritance," because the inheritance of acquired characteristics—Lamarckism—drives the self-appointed evolution police crazy.

ÑóẊîöʼn
22nd August 2011, 04:57
The "switches" themselves would still have had to evolve. I don't see how this is much different from cells differentiating themselves into functional organs and tissues using chemical signaling during embryonic development, but on the level on the organism.

Ocean Seal
22nd August 2011, 05:43
The "switches" themselves would still have had to evolve. I don't see how this is much different from cells differentiating themselves into functional organs and tissues using chemical signaling during embryonic development, but on the level on the organism.
The difference that I see is that those differences aren't passed down through reproduction.

ÑóẊîöʼn
22nd August 2011, 06:28
The difference that I see is that those differences aren't passed down through reproduction.

Well, when stem cells differentiate into liver cells or muscle cells or whatever due to chemical gradients, and those liver and muscle cells continue dividing as liver and muscle cells, I'd say that's a difference passed down through reproduction.

piet11111
22nd August 2011, 08:02
Crocodiles determine their gender through the temperature in the nest so how is this news ?

ÑóẊîöʼn
22nd August 2011, 14:21
A take (http://whyevolutionistrue.wordpress.com/2011/08/21/is-epigenetics-a-revolution-in-evolution/) from evolutionary biologist Jerry Coyne:


Is “epigenetics” a revolution in evolution?

One often hears the suggestion that the neo-Darwinian view of evolution is on the skids, and that that view will be completely changed—if not overturned—by new biological ideas like modularity, genetic assimilation, evolvability, and epigenetics. Epigenetics in particular (I’ll define it in a moment) has been especially touted as a concept that will revolutionize evolutionary biology.

Call me an old fogey, but I think the idea of epigenetics as a Darwin-destroyer is completely bogus. Although certain discoveries in that area are interesting, and have certainly expanded our notion about how genes work, there is not the slightest evidence that the findings of epigenetics will dispel the main ideas of neo-Darwinism, which include the ideas of evolutionary change via natural selection and genetic drift, the randomness of mutations, the ideas of speciation and common descent, and the gene-centered view of evolution. I’ve explained my views on epigenetics as a revolution in several previous posts, for example here (http://whyevolutionistrue.wordpress.com/2010/11/04/epigenetics-kerfuffle/), here (http://whyevolutionistrue.wordpress.com/2010/10/24/epigenetics-the-light-and-the-way/), here (http://whyevolutionistrue.wordpress.com/2010/03/19/worst-science-journalism-of-the-year-darwin-completely-wrong-again/), and here (http://whyevolutionistrue.wordpress.com/2011/07/13/bbc-4-tonight-a-revolution-in-evolution-not/), but, like the Lernean Hydra (http://en.wikipedia.org/wiki/Lernaean_Hydra), each time you cut off a head of the epigenetic beast, it grows another one.

The latest head appeared in Friday’s Guardian, in a book review written by Peter Forbes (http://www.guardian.co.uk/books/2011/aug/19/epigenetics-revolution-nessa-carey-review); the book is The Epigenetics Revolution by Nessa Carey, and Forbes sees the book as tremendously important, implying that is part of a scientific revolution and explicitly saying that it’s a book that would “make Darwin swoon.” I haven’t read the book, and although it might make Darwin swoon if the old git were to be resurrected, the discoveries of genetics and the mechanism of inheritance itself would make him swoon far more readily. And I know scientific revolutions; scientific revolutions are friends of mine; and believe me, epigenetics is no scientific revolution (http://en.wikipedia.org/wiki/Senator,_you%27re_no_Jack_Kennedy£).

So what is epigenetics? The term is actually used in two different ways. When I was younger, it simply meant “developmental genetics,” that is, the study of the way the DNA code of genes is translated into the bodies and physiologies of organisms. That, of course, was a tremendously important and exciting area, and still is. It involves understanding how genes are turned on and off in different tissues and cells, how different genes interact with each other, and how the products of a one-dimensional sequence of information can build a three-dimensional body. This study has segued into the new field of “evo devo,” which tries to understand the evolutionary basis of developmental genetics. “Evo devo” itself has, of course, led to its own important discoveries, like the presence and conservation of homeobox genes, the use of the same genes over and over again in forming similar but non-homologous traits (e.g., PAX6 in the formation of fly eyes and vertebrate eyes), and the linear arrangement of genes in some organisms (e.g., Drosophila) that correspond to the linear arrangement of body parts they affect.

So developmental genetics, and evo devo, are fascinating areas that produce a stream of surprising discoveries. But they’ve done nothing to alter the going paradigm of neo-Darwinian evolution. It is telling that, for example, Sean Carroll, a famous practitioner of “evo devo” and a popular writer, is a firm adherent to neo-Darwinism. What we learn from these areas is precisely how evolution has acted to sculpt bodies, but it still does so using randomly-generated genetic variation and good old natural selection (and yes, Larry Moran, genetic drift also plays a role). Gene regulation itself is a phenomenon molded by natural selection, and how genes are turned on and off is itself a phenomenon residing in the genes: in the genes that make the DNA or proteins that regulate other genes, and in the many ways that genes evolve (through, for example, the evolution of regulatory regions), to respond to internal “environmental” influences.

The second meaning of “epigenetics” is more recent, and involves actual changes in the DNA itself that are not based on mutational changes in nucleotides, but in environmental modifications of nucleotides—things like methylation of nucleotide bases or changes in DNA-associated proteins like histones—that can temporarily modify genes and affect their actions. I say “temporarily,” because such environmental modification of DNA, while it can be adaptive, is not usually passed on from one generation to the next. For example, we get our genes in pairs—one from mom and one from dad—but they can be differentially “marked” (the technical term is “imprinted”) during the formation of sperm and eggs, and so the copy from dad can act differently from the copy coming from mom. This imprinting is probably due to natural selection: scientists like David Haig have argued that the different and conflicting “interests” of paternal versus maternal genes has, through natural selection, molded the way they are imprinted, allowing them to act in different ways in the embryo. But an “imprinted” gene is reset each generation: the imprinting disappears and has to re-form depending on which sex the gene is in.

As I have argued before, however, imprinting of genes, although a novel and recently-discovered phenomenon, is not a “revolution” in how we view evolution: it is an embellishment that doesn’t overturn the main ideas of neo-Darwinism. And many of the phenomena subsumed by this modern notion of “epigenetics” still evolved by natural selection. Imprinting, after all, is based on changes in DNA that somehow render paternal DNA more (or less) susceptible to modification than maternal DNA. Imprinting has evolved by changes in DNA, even though the modifications of DNA it causes are environmental.

In his review of Carey’s book, Forbes, a science writer (http://www.pforbes.org/), concentrates on the second, “revolutionary” sense of epigenetics:

Genes don’t just issue instructions: they respond to messages coming from other genes, from hormones and from nutritional cues and learning. Much epigenetics revolves around nutrition. If we drink a lot of alcohol an enzyme that metabolises it becomes more active – “upregulated” in the jargon. And similar mechanisms apply to much of our behaviour. The methods by which genes makes these responses often involve very small chemical modifications (usually the addition of a tiny methyl group to one base of DNA). It is almost certain that memory – a classic nurture problem: we learn something and it becomes biologically encoded – involves epigenetics. Once made, epigenetic changes can be very long lasting, which is how our long-term memory is possible.

Why is this “revolutionary?” Because some of the inherited changes of genes appear to be “Lamarckian,” that is, they aren’t really changes in DNA sequence itself, but environmental modifications of DNA that can be passed from one generation to the next. And if such “nongenetic,” environmentally-acquired inheritance were common, that would be a revolution in the way we think about evolution.

So what’s the evidence for this “revolutionary” notion? Forbes simply offers up the same tired old anecdotes I’ve addressed before:

So far, this is instructive and highly promising for medical research, but epigenetics finally reaches that “everything you’ve been told is wrong” moment when it claims that some epigenetic changes are so long-lasting they cover several generations: they can be inherited. This flouts one of biology’s most cherished dogmas – taught to all students – namely that changes acquired during life cannot be passed on – the heresy of Lamarckism.

But the evidence that this can occur in some cases appears to be growing. There are lab experiments with mice and rats in which epigenetic effects on coat colour and obesity can be inherited. More suggestive evidence comes from a vast, unwitting and cruel experiment played out in the second world war. In 1944, during the last months of the war, a Nazi blockade followed by an exceedingly harsh winter led to mass starvation in Holland. This had a huge effect on babies born at the time, and the effects of poor nutrition on the foetus seem to have persisted through subsequent generations.

Well, I won’t flog poor Mr. Forbes with the fact that these are only a few trivial examples of the phenomenon, examples that don’t appear to have any evolutionary importance. Nor will I flog him with the fact that when we can dissect the genetic basis of real adaptations in real organisms, they invariably turn out to rest on changes in DNA sequence, not in environmental and non-DNA-based modifications of nucleotides. Here’s what I said in an earlier post (http://whyevolutionistrue.wordpress.com/2010/03/19/worst-science-journalism-of-the-year-darwin-completely-wrong-again/) about Oliver Burkeman’s claims that epigenetics has profound implications for evolution (like Forbes, Burkeman is a science journalist):

All I can say to this is: “Profound implications my tuchus!” There are a handful of examples showing that environmentally-induced changes can be passed from one generation to the next. In nearly all of these examples, the changes disappear after one or two generations, so they couldn’t effect permanent evolutionary change. The proponents of epigenesis as an important factor in evolution, like Eva Jablonka and Marion Lamb, always wind up talking about the same tired old examples, like cases of coat color change in mice and flower pattern in toadflax (http://www.nature.com/nature/journal/v447/n7143/full/nature05913.html). I am not aware of a single case in which an adaptive change in an organism—or any change that has been fixed in a species—rests on inheritance that is not based on changes in the DNA. (For a refutation of the pro-epigenesis arguments that Jablonka and Lamb make in their 2005 book, see Haig [2007].)

Moreover, some examples of “nongenetic” inheritance that do have adaptive significance, such as differential methylation of paternal versus maternal chromosomes, ultimately rest on changes in DNA that promote that differential methylation. And this “inheritance” lasts only one generation, for the methylation profile is reset in each sex every generation.

In contrast to the very few cases of one- or two-generation inheritance that cause nonadaptive changes in the phenotype stands the very, very large number of studies in which inherited changes within and among species map to the DNA. These include every case of evolutionary response to artificial or human-generated selection, adaptive changes within species (e.g., spiny-ness in sticklebacks), and differences among species in both morphology (e.g., the color differences in fruit flies I study) and reproductive barriers (the many mapping studies of “hybrid sterility” and “hybrid inviability” genes). Burkeman, of course, doesn’t mention these cases: it would ruin his nice story.

If we look just at studies of the inheritance of organismal changes that have evolved over time (and many of these would have detected profound epigenetic effects), the score would be something like this: DNA 757, Epigenesis 0. (I’m just making these numbers up, of course, to make a point.) If we look at all “inherited changes”, regardless of their evolutionary importance, we would have a handful of epigenetic changes versus literally thousands of DNA-based changes. So how can Burkeman say that epigenesis will profoundly revise our view of evolution?

So, Mr. Forbes, our “cherished dogma” of non-Lamarckian inheritance still holds strong, and you’ve done your readers a disservice by implying otherwise. Lamarckism is not a “heresy,” but simply a hypothesis that hasn’t held up, despite legions of evolution-revolutionaries who argue that it flushes neo-Darwinism down the toilet. If “epigenetics” in the second sense is so important in evolution, let us have a list of, say, a hundred adaptations of organisms that evolved in this Larmackian way as opposed to the old, boring, neo-Darwinian way involving inherited changes in DNA sequence.

Forbes can’t produce such a list, because there’s not one. In fact, I can’t think of a single entry for that list.

Science journalists—meh. They’re always trying to argue that Darwin was wrong and that evolution is about to undergo a Kuhnian revolutionary paradigm. But what they really want is readership, and you don’t get readers by writing that the conventional wisdom happens to be correct.

Lynx
22nd August 2011, 14:49
Some mutations may be pseudo-random, some are recurring, the rest are likely triggered.

Epigenetics is not Lamarckism!